Corrigendum to "Fatty acid oxidase Ehhadh mediates stem cell fate remodeling via mitophagy activation" [Free Radic. Biol. Med. 248 (2026) 109-126] [0.03%]
关于“脂肪酸氧化酶Ehhadh通过激活线粒体自噬介导干细胞命运重塑”的勘误[自由基生物医学杂志248（2026）109-126]
Jingxuan Zhou,Longyun Zhang,Tingjun Liu et al.
Jingxuan Zhou et al.
HDL/ApoA1 attenuates atherosclerosis by suppressing macrophage ferroptosis via NRF2-SLC7A11-GSH axis activation [0.03%]
apoA1通过激活NRF2-SLC7A11-GSH轴抑制巨噬细胞铁死亡从而发挥抗动脉粥样硬化作用
Yuting Yi,Yonghong Luo,Yongjie Deng et al.
Yuting Yi et al.
Background: Macrophage death modalities critically determine atherosclerotic plaque stability and progression. While high-density lipoprotein (HDL) and its major component apolipoprotein A1 (ApoA1) are known for their cho...
Salvianolic acid B mitigates neuronal ferroptosis after intracerebral hemorrhage in rats through a Piezo1-associated AMPK-mTOR pathway [0.03%]
天冬酸二乙酯通过Piezo1相关的AMPK-mTOR信号通路减轻出血性卒中后神经元铁死亡作用机制研究
Zhongyi Zhang,Linhui Zhang,Ye Yuan et al.
Zhongyi Zhang et al.
Secondary brain injury (SBI) after intracerebral hemorrhage (ICH) is driven in part by iron-dependent oxidative damage and neuronal ferroptosis, but the molecular signals associated with ferroptotic vulnerability in the hemorrhagic brain re...
Myeloid specific knockout of Piezo1 alleviates ang Ⅱ-induced cardiac remodeling [0.03%]
髓系细胞特异性Piezo1敲除缓解AngⅡ诱导的心脏重塑
Jiali Liu,Weipin Niu,Pangao Xu et al.
Jiali Liu et al.
Introduction: Cardiac remodeling in heart failure is closely associated with macrophage-mediated immune responses. The mechanosensitive ion channel Piezo1, which is abundantly expressed in macrophages, has been implicated...
Microglia-derived exosomal miR-31-5p promotes type 2 diabetic retinopathy by impairing physiological angiogenesis homeostasis [0.03%]
小胶质细胞来源的外泌体miR-31-5p通过破坏生理血管内稳态促进糖尿病视网膜病变
Jie Zhang,Yanyu Shangguan,Ruoning Luo et al.
Jie Zhang et al.
Diabetic retinopathy (DR) is characterized by disruption of the retinal physiological vasculature. Impairment of physiological angiogenesis profoundly destabilizes the retinal microenvironment and ultimately leads to visual dysfunction. Cur...
Shuang Yang,Chengye Zhang,Zhusong Mei et al.
Shuang Yang et al.
Cold tumors, defined by an immunosuppressive microenvironment and metabolic stress, including glutamine deficiency, frequently exhibit resistance to therapeutic interventions. This study examined the role of glutathione peroxidase 1 (GPX1) ...
Frataxin attenuates endothelial inflammation triggered by engulfment of senescent erythrocytes [0.03%]
血红蛋白吞噬诱发内皮细胞炎症反应的负调控因子-frataxin蛋白质
Xingyi Cheng,Yi Cheng,Chenglin Huang et al.
Xingyi Cheng et al.
Background: Hypertension is linked to a shortened erythrocyte lifespan, with endothelial cells acting as non-professional phagocytes to clear senescent erythrocytes. However, whether increased erythrophagocytosis contribu...
An enzyme kinetic model for quantitative interpretation of the role of nicotinamide nucleotide transhydrogenase (NNT) in cell physiology [0.03%]
烟酰胺核苷酸转氢酶(NNT)在细胞生理中的作用的定量解释的酶动力学模型
Zhuohui Gan,Inge van der Stelt,Sander Grefte et al.
Zhuohui Gan et al.
Nicotinamide Nucleotide Transhydrogenase (NNT) connects mitochondrial bioenergetics and redox homeostasis. It catalyzes a reversible reaction that couples proton transport across the mitochondrial inner membrane (MIM) to hydride transfer be...
Corrigendum to "The opposite role of lactate dehydrogenase a (LDHA) in cervical cancer under energy stress conditions" [Free Radic. Biol. Med. 214 (2024) 2-18] [0.03%]
关于“乳酸脱氢酶a(LDHA)在宫颈癌能量应激条件下的相反作用”的勘误说明[自由基生物学与医学杂志，第214期(2024年)2-18页]
Chaoran Jia,Yulun Wu,Feng Gao et al.
Chaoran Jia et al.
Complement C3a induces synaptic elimination in the hippocampus and neurobehavioral deficits in later life of septic neonatal rats triggered by lipopolysaccharide [0.03%]
脂多糖诱发的新生大鼠败血症中补体C3a诱导海马区突触丢失并导致成年后出现神经行为缺陷
Peixian Huang,Shuqi Jiang,Zhuo Chen et al.
Peixian Huang et al.
Neonatal septic encephalopathy is a severe complication of sepsis, yet its underlying neuropathological mechanisms remain poorly understood. This study investigated the role of the complement component C3a in mediating synaptic elimination ...