Should we stimulate or suppress immune responses in COVID-19? Cytokine and anti-cytokine interventions [0.03%]
应刺激还是抑制新冠肺炎患者的免疫反应？细胞因子和抗细胞因子干预疗法
Yvan Jamilloux,Thomas Henry,Alexandre Belot et al.
Yvan Jamilloux et al.
The coronavirus disease-19 pandemic (COVID-19), which appeared in China in December 2019 and rapidly spread throughout the world, has forced clinicians and scientists to take up extraordinary challenges. This unprecedented situation led to ...
Zhao-Wei Gao,Xi Wang,Fang Lin et al.
Zhao-Wei Gao et al.
SARS-CoV-2 infection complicated by inflammatory syndrome. Could high-dose human immunoglobulin for intravenous use (IVIG) be beneficial? [0.03%]
SARS-CoV-2感染合并炎症综合征高剂量人免疫球蛋白静脉给药能否获益？
Marcella Prete,Elvira Favoino,Giacomo Catacchio et al.
Marcella Prete et al.
Molecular mimicry between SARS-CoV-2 and respiratory pacemaker neurons [0.03%]
SARS-CoV-2与呼吸起搏神经元之间的分子模拟关系
Guglielmo Lucchese,Agnes Flöel
Guglielmo Lucchese
Re: Patiently waiting for the results of anti-IL 6 therapy in severe COVID-19 infection [0.03%]
关于等待抗IL-6治疗重症新冠肺炎的结果的回复
Dennis McGonagle,Kassem Sharif,Anthony O&#x;Regan et al.
Dennis McGonagle et al.
Corona (COVID-19) time musings: Our involvement in COVID-19 pathogenesis, diagnosis, treatment and vaccine planning [0.03%]
冠状病毒（COVID-19）时期思考：我们在新冠肺炎发病学、诊断、治疗和疫苗计划中的作用
Yehuda Shoenfeld
Yehuda Shoenfeld
ACE2 at the centre of COVID-19 from paucisymptomatic infections to severe pneumonia [0.03%]
ACE2在从轻微症状感染到严重肺炎的COVID-19发病过程中的中心地位
Felice Rivellese,Edoardo Prediletto
Felice Rivellese
The Role of Cytokines including Interleukin-6 in COVID-19 induced Pneumonia and Macrophage Activation Syndrome-Like Disease [0.03%]
细胞因子在新冠肺炎和类噬血细胞性淋巴组织细胞增多症中的作用包括白细胞介素-6的作用
Dennis McGonagle,Kassem Sharif,Anthony O&#x;Regan et al.
Dennis McGonagle et al.
Severe COVID-19 associated pneumonia patients may exhibit features of systemic hyper-inflammation designated under the umbrella term of macrophage activation syndrome (MAS) or cytokine storm, also known as secondary haemophagocytic lymphohi...
Daniela Marotto,Piercarlo Sarzi-Puttini
Daniela Marotto
A Unifying Hypothesis of the Pathophysiology of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): Recognitions from the finding of autoantibodies against ß2-adrenergic receptors [0.03%]
肌痛性脑脊髓炎/慢性疲劳综合征(ME/CFS)发病机制统一假说的认识：β2肾上腺素受体自身抗体的发现给予我们的启示
Klaus Wirth,Carmen Scheibenbogen
Klaus Wirth
Myalgic Encephalomyelitis or Chronic Fatigue Syndrome (CFS/ME) is a complex and severely disabling disease with a prevalence of 0.3% and no approved treatment and therefore a very high medical need. Following an infectious onset patients su...