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期刊名:Journal of inflammation-london

缩写:J INFLAMM-LOND

ISSN:1476-9255

e-ISSN:1476-9255

IF/分区:5.8/Q1

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共收录本刊相关文章索引811
Clinical Trial Case Reports Meta-Analysis RCT Review Systematic Review
Classical Article Case Reports Clinical Study Clinical Trial Clinical Trial Protocol Comment Comparative Study Editorial Guideline Letter Meta-Analysis Multicenter Study Observational Study Randomized Controlled Trial Review Systematic Review
G Rinaldi,K K W Cheng,C McCann et al. G Rinaldi et al.
Several cellular pathways lead to the formation of neutrophil extracellular traps (NETs), a form of cell death (NETosis), distinct from apoptotic and necrotic cell death. Surprisingly, there remains a paucity of methods enabling efficient q...
Daniel C Shippy,Abigail H Evered,Tyler K Ulland Daniel C Shippy
Background: Gout arthritis (GA) flares are unexpected bouts of heat, swelling, and redness resulting in excruciating pain caused by monosodium urate (MSU) crystal deposition in the synovial joints. GA flare symptoms occur...
Marco De Zuani,Petra Lázničková,Marcela Hortová Kohoutková et al. Marco De Zuani et al.
Background: Sepsis is a life-threatening condition characterised by an overwhelming immune response and high fatality. While most research has focused on its acute phase, many sepsis survivors remain immunologically weake...
Meng Wang,Weipeng Song,Xiaoming Liu et al. Meng Wang et al.
Background: Chronic obstructive pulmonary disease (COPD) is a significant global cause of morbidity and mortality and is characterized by airway remodeling, inflammation, and endoplasmic reticulum stress (ER stress) progr...
Sarah E Moreno,Joseph Mikulin,Michelle Massee et al. Sarah E Moreno et al.
Background: The immune system plays a pivotal role in progressing an injury through the healing cascade. However, comorbidities often lead to dysregulation of this response and are implicated in wound chronicity or stalli...
Yunliang Liu,Yuting Huo,Chaofeng Liu et al. Yunliang Liu et al.
Objective: This study aimed to investigate the regulation of allergic rhinitis (AR) by methyltransferase 3 through an m6A-dependent mechanism, providing a theoretical foundation for its treatment. ...