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期刊名:Carcinogenesis

缩写:CARCINOGENESIS

ISSN:0143-3334

e-ISSN:1460-2180

IF/分区:2.9/Q2

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共收录本刊相关文章索引3717
Clinical Trial Case Reports Meta-Analysis RCT Review Systematic Review
Classical Article Case Reports Clinical Study Clinical Trial Clinical Trial Protocol Comment Comparative Study Editorial Guideline Letter Meta-Analysis Multicenter Study Observational Study Randomized Controlled Trial Review Systematic Review
Jonica Campolo,Giuseppe Annoni,Andrea Borghini et al. Jonica Campolo et al.
The risk of cancer from medical exposure to ionizing radiation (IR) in pediatric patients-particularly those with congenital heart disease (CHD)-is a concern, given the need for repeated X-ray-guided cardiac catheterization (CC) procedures ...
Breanne Freeman,Tengfei Bian,Gujie Xu et al. Breanne Freeman et al.
4-(Methyl-nitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and its major metabolite 4-(methyl-nitrosamino)-1-(3-pyridyl)-1-butanol (NNAL) are tobacco-specific lung carcinogens. NNK is widely used in preclinical models to induce lung tumorigenesi...
Jaylon Aggison,Cristian Medina,Siqi Wu et al. Jaylon Aggison et al.
Pleural mesothelioma (PM) usually occurs many years after asbestos fiber exposure; yet the mechanisms that convert chronic damage into malignancy remain unclear. Asbestos fibers induce persistent oxidative and genomic stress that should act...
Yi Liu,Shuai Li,Wei Li et al. Yi Liu et al.
Tumor-associated macrophages (TAMs) are the core innate immune cells in the tumor microenvironment (TME), and their phenotypic polarization and functional reprogramming determine the orientation of the tumor immune microenvironment and the ...
Masaaki Sunaoshi,Erika Takahashi,Yoshiko Amasaki et al. Masaaki Sunaoshi et al.
High linear energy transfer (LET) radiation is thought to induce highly complex DNA damage compared with photon radiation such as X-rays, suggesting a characteristic mechanism for the development of cancers associated with high-LET radiatio...
Kun-Ming Chen,Nicolle M Krebs,Yuan-Wan Sun et al. Kun-Ming Chen et al.
The tobacco constituent, N'-nitrosonornicotine (NNN) is a potential etiological agent in the development of oral cancer. NNN is metabolically activated to form DNA adducts that release 4-hydroxy-1-(3-pyridyl)-1-butanone (HPB) which was dete...