Corrigendum to 'FABP4, marker of worse prognosis in cardiovascular disease, induces neutrophil's proatherogenic phenotype which is modulated by semaglutide' [Journal of Molecular and Cellular Cardiology volume 210 (2026) 12-27] [0.03%]
关于“FABP4是心血管疾病预后较差的标志物，诱导中性粒细胞促动脉粥样化的表型并被索马鲁肽调节”（《分子和细胞心脏病学杂志》第210卷(2026) 12-27页）的勘误通知书
David Sánchez-López,David García-Vega,J E Viñuela et al.
David Sánchez-López et al.
ZIP14 upregulation leads to ferroptosis and lysosomal dysfunction through intracellular iron overload and induces myocardial ischemia/reperfusion injury in mouse hearts [0.03%]
ZIP14上调通过细胞内铁过载诱导铁死亡和溶酶体功能障碍并导致小鼠心脏缺血再灌注损伤
Liang Zhao,Yuru Cao,Xianle Liu et al.
Liang Zhao et al.
While the ZIP family Zn2+ transporters such as ZIP2 and ZIP7 play critical roles in myocardial ischemia/reperfusion (I/R) injury by regulating Zn2+ homeostasis, little is known about the roles of the other ZIP family Zn2+ transporters in I/...
Cyclic AMP-dependent regulation of ryanodine receptors in healthy and diseased hearts [0.03%]
环AMP依赖的调控在健康和患病心脏中的雷诺丁受体调节中发挥作用
Mohit M Hulsurkar,Isabelle Ong,Joshua A Keefe et al.
Mohit M Hulsurkar et al.
Cyclic adenosine monophosphate (cAMP) is a critical second messenger in cardiomyocytes, regulating essential cellular functions. Upon G-protein-coupled receptor stimulation, adenylyl cyclase (AC) synthesizes cAMP, which phosphodiesterase (P...
Impaired retinoic acid signaling mediated Rbm20 downregulation induces aberrant splicing of CaV1.2 calcium channel: implications in myocardial hypertrophy [0.03%]
视黄酸信号受损介导的Rbm20下调引起CaV1.2钙通道异常剪接：在心肌肥厚中的意义
Xinyu Song,Dan Yang,Zhe Sun et al.
Xinyu Song et al.
Aims: Myocardial hypertrophy, a pathological adaptation to chronic stress, predisposes to heart failure through dysregulated calcium handling. Alternative splicing (AS) of CaV1.2 calcium channel participates in myocardial...
Shivani Sethi,Isaiah Cheong,Carol T Bussey et al.
Shivani Sethi et al.
Diabetic heart disease is a leading cause of morbidity and mortality in individuals with type 2 diabetes mellitus (T2DM). A major yet frequently under-recognized component of diabetic heart disease is cardiac autonomic neuropathy (CAN), a c...
Sphingosine-1-phosphate activates ICl,swell in ventricular myocytes via mitochondrial reactive oxygen production [0.03%]
鞘氨醇-1-磷酸通过线粒体活性氧生成激活心室肌细胞的ICl,swell
Frank J Raucci,Adolfo G Mauro,Edward J Lesnefsky et al.
Frank J Raucci et al.
Aims: We previously demonstrated that bacterial sphingomyelinase (SMase), which converts plasmalemmal sphingomyelin to long-chain ceramides, activates the swelling-activated chloride current (ICl,swell) in rabbit ventricu...
Interplay between variability in intrinsic cellular properties and heart failure-associated remodeling in a simulated population with human heart failure [0.03%]
含有人心力衰竭患者的固有细胞特异性的变异性和与心力衰竭相关的心肌重构的相互作用
Jacob A Miller,Nicolae Moise,Mario J Mendez et al.
Jacob A Miller et al.
Heart failure (HF) is the presentation of mechanical pump dysfunction, with HF patients facing increased risk of sudden cardiac death predominantly driven by ventricular arrhythmias. At the cellular level, HF is associated with remodeling o...
Spatiotemporal determinants of stretch-activated channel-induced re-entry in ventricular tissue: An in-silico study [0.03%]
心室组织中伸缩激活通道诱导再激动的时空决定因素：一项仿真研究
Melania Buonocunto,Tammo Delhaas,Aurore Lyon et al.
Melania Buonocunto et al.
Stretch-activated ion channels (SACs) mediate mechano-electric feedback in cardiomyocytes by coupling mechanical and electrical activity. While SACs activation can induce pro-arrhythmic effects at the cellular level, its impact on tissue-le...
PhysioMEA: Signal processing platform for rate and rhythm analysis of multi-electrode array cardiac electrophysiological recordings [0.03%]
基于多电极阵列心脏电生理记录的心率和心律分析信号处理平台
Ido Weiser-Bitoun,Savyon Mazgaoker,Shani Assayag et al.
Ido Weiser-Bitoun et al.
Cardiac organoids serve as a valuable model for studying physiological and pathophysiological processes affecting heart rate and rhythm. Multi-electrode arrays (MEA) are widely used for high-throughput electrophysiological assessments. Desp...