[Clinical Significance of SFRP1 Gene Methylation in Patients with Childhood Acute Lymphoblastic Leukemia] [0.03%]
儿童急性淋巴细胞白血病患者中SFRP1基因甲基化的临床意义
Jing Yan,Wen-Peng Wang,Xuan Li et al.
Jing Yan et al.
Objective: To investigate the clinical significance of SFRP1 gene and its methylation in childhood acute lymphoblastic leukemia (ALL) . Methods: ...
[The Effects and Regulatory Mechanism of Targeting CXC Chemokine Receptor 1/2 Combined with Ara-C on the Malignant Biological Behaviors of U937 Cells of Acute Myeloid Leukemia] [0.03%]
靶向CXCR1/CXCR2联合Ara-C对急性髓系白血病U937细胞恶性生物学行为的影响及机制研究
Yan-Quan Liu,Jian-Zhen Shen,Yue Yin et al.
Yan-Quan Liu et al.
Objective: To investigate and analyze the effect of CXC chemokine receptor 1/2 (CXCR1/2) targeting inhibitor Reparixin combined with cytarabine (Ara-C) on the malignant biological behaviors of acute myeloid leukemia cells...
[Effect of Scutellarin on Proliferation of Acute Myeloid Leukemia Cells and Its Related Mechanism] [0.03%]
鼠尾草酚对急性髓系白血病细胞增殖的影响及其作用机制研究
Jun Chen,Yi-Feng Cai,Min Shao et al.
Jun Chen et al.
Objective: To investigate the effect of scutellarin (SCU) on proliferation, cell cycle and apoptosis of acute myeloid leukemia (AML) cells and its related molecular mechanism. ...
[Clinical Analysis of SET-NUP214 Fusion Gene Positive Patients with Acute Leukemia] [0.03%]
SET-NUP214融合基因阳性急性 leukemia患者的临床分析
Yang Song,Xiao-Yuan Gong,Shu-Ning Wei et al.
Yang Song et al.
Objective: To analyze the characteristics and prognosis of acute leukemia(AL) with SET-NUP214 fusion gene. Methods: The clinical data o...
[The Mechanism of miR-1294 Targeting SOX15 to Regulate Wnt/β-catenin Signaling Pathway and Promote the Proliferation of Acute Lymphoblastic Leukemia Cells in Children] [0.03%]
miR-1294通过靶向SOX15调节Wnt/β-catenin信号通路促进儿童急性淋巴细胞白血病细胞增殖的机制研究
Hong-Xia Cen,Si-Ming Cai,Hong-Yu Jiang et al.
Hong-Xia Cen et al.
Objective: To explore the effect of abnormal miRNA expression on the proliferation of pediatric acute lymphoblastic leukemia (ALL) cells and its related mechanism. ...
[Efficacy and Safety of Decitabine Combined with Modified EIAG Regimen in the Treatment of Patients with Relapsed/Refractory Acute Myeloid Leukemia and High-risk Myelodysplastic Syndrome] [0.03%]
地西他滨联合改良DAE方案治疗复发难治急性髓系白血病及高危骨髓增生异常综合征患者的疗效及安全性分析
Jian-Ping Mao,Lian-Guo Xue,Yuan-Xin Zhu et al.
Jian-Ping Mao et al.
Objective: To investigate the efficacy, prognosis and safety of decitabine combined with modified EIAG regimen in the treatment of patients with relapsed/refractory acute myeloid leukemia (AML) and high-risk myelodysplast...
[Correlation between ARID5B Gene SNP and MTX Resistance in Children with ALL] [0.03%]
ARID5B基因多态性与儿童急性淋巴细胞白血病甲氨蝶呤耐药的相关性研究
Li-Fen Zhang,Yu Ma,Lian Li et al.
Li-Fen Zhang et al.
Objective: To investigate the correlation between single-nucleotide polymorphism (SNP) of ARID5B gene and resistance to methotrexate (MTX) in children with acute lymphoblastic leukemia (ALL). ...
[Clinical Observation of Venetoclax Combined with Demethylating Agents on the Treatment of Relapsed/Refractory Acute Myeloid Leukemia] [0.03%]
伏立诺他联合地西他滨治疗复发难治急性髓系白血病的临床观察
Yao Wang,Sai-Lan Huang,Xing-Xia Zhang et al.
Yao Wang et al.
Objective: To investigate the efficacy and safety of venetoclax (VEN) combined with demethylating agents (HMA) in the treatment of relapsed/refractory acute myeloid leukemia (R/R AML). ...
[Inhibitory Effect of Kaempferol on Proliferation of KG1a Cells and Its Mechanism] [0.03%]
山柰酚对白血病KG1a细胞增殖的抑制作用及机制研究
Zhe Chen,Ling Zhang,Xiao-Fei Yuan et al.
Zhe Chen et al.
Objective: To investigate the effect of kaempferol on proliferation of acute myeloid leukemia (AML) KG1a cells and its mechanism. Methods: ...
[Establishment of a Patient-Derived T-Cell Acute Lymphoblastic Leukemia Xenograft Model in Novel Immunodeficient NCG Mice] [0.03%]
[NCG小鼠人T细胞急性淋巴细胞白血病患者源性异种移植模型的建立]
Peng-Jun Jiang,Xing-Bin Dai,Xiang-Tu Kong et al.
Peng-Jun Jiang et al.
Objective: The leukemia cells from patients with T-cell acute lymphoblastic leukemia (T-ALL) were inoculated into NCG mice to establish a stable human T-ALL leukemia animal model. ...