Correction to "Controlled Hemorrhage Sensitizes Angiotensin II-Elicited Hypertension Through Activation of the Brain Renin-Angiotensin System Independently of Endoplasmic Reticulum Stress" [0.03%]
“受控失血通过激活大脑肾素血管紧张素系统独立于内质网应激而使血管紧张素Ⅱ诱导的高血压易感化”的勘误通知
[This corrects the article DOI: 10.1155/2022/6371048.]. Copyright © 2026 Oxidative Medicine and Cellular Longevity published by John Wiley & Sons Ltd.
Published Erratum
Oxidative medicine and cellular longevity. 2026 Jan 7:2026:9829207. DOI:10.1155/omcl/9829207 2026
RETRACTION: Sesamin Enhances Nrf2-Mediated Protective Defense against Oxidative Stress and Inflammation in Colitis via AKT and ERK Activation [0.03%]
撤回：sesamin通过活化AKT和ERK增强Nrf2介导的防御作用以抵抗结肠炎中的氧化应激和炎症
Oxidative Medicine And Cellular Longevity
Oxidative Medicine And Cellular Longevity
[This retracts the article DOI: 10.1155/2019/2432416.]. Copyright © 2025 Oxidative Medicine and Cellular Longevity published by John Wiley & Sons Ltd.
Sotagliflozin Modulation of SIRT1/Nrf2 and PI3K/AKT Signaling Pathway Ameliorates Experimental Liver Fibrosis in Rats [0.03%]
Sotagliflozin通过调节SIRT1/Nrf2和PI3K/Akt信号通路改善大鼠实验性肝纤维化
Hossein M Elbadawy,Mohannad A Almikhlafi,Mohammed H Alsubhi et al.
Hossein M Elbadawy et al.
Background and purpose: Liver fibrosis poses a major global health burden, contributing substantially to morbidity and mortality worldwide. This study aims to assess the potential novel mechanisms behind the anti-fibrotic...
Coffee Wastes: A Sustainable Source of Natural Compounds Suppressing Colorectal Cancer Cell Viability [0.03%]
咖啡废料：一种可持续的天然化合物抑制结直肠癌细胞活力的来源
Mariavittoria Verrillo,Paola Cuomo,Cristina Pagano et al.
Mariavittoria Verrillo et al.
Colorectal cancer (CRC) is one of the leading causes of cancer-related deaths worldwide. Emerging evidence suggests a rising incidence of CRC in younger adults, emphasizing the urgent need for innovative therapeutic strategies. The increasi...
RETRACTION: Oxidative Stress and Pulmonary Changes in Experimental Liver Cirrhosis [0.03%]
撤稿：实验性肝硬化中的氧化应激和肺部变化
Oxidative Medicine And Cellular Longevity
Oxidative Medicine And Cellular Longevity
[This retracts the article DOI: 10.1155/2012/486190.]. Copyright © 2025 Oxidative Medicine and Cellular Longevity published by John Wiley & Sons Ltd.
Correction to "Calorie Restriction Protects against Contrast-Induced Nephropathy via SIRT1/GPX4 Activation" [0.03%]
关于“ calorie restriction通过SIRT1/GPX4激活保护免受对比剂引起的肾病”的更正通知
[This corrects the article DOI: 10.1155/2021/2999296.]. Copyright © 2025 Oxidative Medicine and Cellular Longevity published by John Wiley & Sons Ltd.
Targeting NRF2 With Isoeugenol: A Promising Small Molecule for Neurodegenerative, Metabolic, and Chronic Inflammatory Disorders [0.03%]
异丁香酚靶向NRF2：一种治疗神经退行性、代谢和慢性炎症疾病的前景小分子
Ana Silva,Sónia Silva,Beatriz Rodrigues et al.
Ana Silva et al.
Oxidative stress, driven by an imbalance between oxidants and antioxidants, disrupts redox homeostasis and contributes to the development of chronic diseases, including cancer, diabetes, neurodegenerative disorders, and aging. The NRF2-KEAP...
Targeting NRF2 With Isoeugenol: A Promising Small Molecule for Neurodegenerative, Metabolic, and Chronic Inflammatory Disorders [0.03%]
异丁香酚靶向NRF2：一种针对神经退行性、代谢和慢性炎症障碍的有前景的小分子
Ana Silva,Sónia Silva,Beatriz Rodrigues et al.
Ana Silva et al.
Oxidative stress, driven by an imbalance between oxidants and antioxidants, disrupts redox homeostasis and contributes to the development of chronic diseases, including cancer, diabetes, neurodegenerative disorders, and aging. The NRF2-KEAP...
Detecting Glutathione and Related Antioxidants as Biomarkers in Patient Breast Tumor Tissues: An Update in the Age of Metabolomics [0.03%]
代谢组学时代的谷胱甘肽和相关抗氧化剂作为患者乳腺肿瘤组织生物标志物的检测进展情况
Michael P Gamcsik
Michael P Gamcsik
Increased levels of glutathione (GSH) and related antioxidant processes are thought to predict breast tumor aggressiveness and therapy response. In our 2012 review of 21 studies, we found that most patient breast tumors exhibited increased ...
Correction to " N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway Activation Induced by Helicobacter pylori Infection" [0.03%]
关于“N-乙酰半胱氨酸减少Helicobacter pylori感染诱导的ROS介导的氧化性DNA损伤及PI3K／Akt途径活化”的更正通知
[This corrects the article DOI: 10.1155/2018/1874985.]. Copyright © 2025 Oxidative Medicine and Cellular Longevity published by John Wiley & Sons Ltd.