Motor neuron disease and serum monoclonal proteins: poor response to treatment of the paraproteinemia [0.03%]
肌萎缩侧索硬化伴单克隆免疫蛋白血症及其疗效不良的一项回顾性研究
M T Burns,V C Juel,C E Hess et al.
M T Burns et al.
An association between motor neuron disease (MND) and paraproteinemia has been previously reported. In a retrospective study, we found that 13 of 117 (11%) patients with MND who had serum protein electrophoresis studies were found to have a...
S B Rutrove,R A Nardin,E M Raynor et al.
S B Rutrove et al.
Objective: To determine the relative importance of lumbosacral polyradiculopathy (PR) as a cause of distal lower extremity sensory loss, weakness, and hyporeflexia usually attributed to polyneuropathy (PN). METHODS.: We p...
Tomacufous neuropathies [0.03%]
类肉瘤神经病
S S Jaradeh,S K Danielson
S S Jaradeh
H Halford,A Graves,T Bertorini
H Halford
Imaging of muscle and nerve has become increasingly useful and of promising value in the evaluation of patients with various neuromuscular disorders. These techniques include ultrasonography, radionuclide scanning, computed tomography, and ...
Myositis-specific and -associated autoantibodies: a review from the clinical perspective [0.03%]
肌炎特异性和关联性自身抗体：临床视角的综述
D Lacomis,C V Oddis
D Lacomis
Twenty-five percent to 50% of patients with polymyositis (PM) and dermatomyositis (DM) have autoantibodies that are specific for or associated with the presence of myositis. Because of a relatively low sensitivity for the diagnosis of PM or...
What's in the Literature? [0.03%]
文献述评
L H Phillips nd
L H Phillips nd
Late-Onset Friedreich's Ataxia Presenting as a Spastic Paraparesis [0.03%]
晚发弗里德赖希共济失调以痉挛性轻偏瘫为首发症状的1例报告
D S Silvers,K J Felice
D S Silvers
A 55-year-old woman presented with a 10-yeai history of a progressive gait disorder. Her examination showed a spastic paraparesis with brisk deep tendon reflexes, but only minimal limb ataxia and no evidence for a sensory neuropathy The pat...
A Melberg,R Raininko,N Dahl et al.
A Melberg et al.
We report on, 1 36-year-old man of Swedish descent who had teenage onset of a progressive disorder with features of Charcot-Marie-Tooth disease (CMT) and cerebellar ataxia. Cognition was normal. The polyneuropathy was of axonal type. Magnet...