Corrigendum to "The combination therapy using tyrosine kinase receptors inhibitors and repurposed drugs to target patient-derived glioblastoma stem cells" [Biomed. Pharmacother. 176 (2024) 116892] [0.03%]
关于“使用酪氨酸激酶受体抑制剂和老药联合疗法靶向患者来源的胶质母细胞瘤干细胞”一文的勘误[Biomed. Pharmacother. 176 (2024) 116892]
Malgorzata Kucinska,Julia Pospieszna,Jing Tang et al.
Malgorzata Kucinska et al.
A Novel Class of Multi-substituted Diaryl Scaffold Derivatives Inhibit Glioblastoma Progression by Targeting CD155 [0.03%]
一类多取代二芳基支架衍生物通过靶向CD155抑制胶质母细胞瘤进展
Yong-Jian Wang,Ting Sun,Si-Tu Xue et al.
Yong-Jian Wang et al.
The immune targeting of glioblastoma stem cells (GSCs) holds great promise.
FGFR inhibition as a new therapeutic strategy to sensitize glioblastoma stem cells to tumor treating fields [0.03%]
FGFR抑制作为一种新的治疗策略用于使胶质母细胞瘤干细胞对肿瘤治疗电场敏感化
Pauline Deshors,Ziad Kheil,Laetitia Ligat et al.
Pauline Deshors et al.
Glioblastomas (GBM) are aggressive tumors, which systematically relapse despite standard treatment associating surgery, chemotherapy and radiation therapy. More recently, GBM therapy now includes another therapeutic modality option, Tumor T...
Importin-alpha transports Norrin to the nucleus to promote proliferation and Notch signaling in glioblastoma stem cells [0.03%]
进口蛋白α通过将Norrin转运至细胞核来促进胶质母细胞瘤干细胞的增殖和Notch信号传导
Ahmed El-Sehemy,Nobuhiko Tachibana,Arturo Ortin-Martinez et al.
Ahmed El-Sehemy et al.
Subsequently, we demonstrate that KPNA2 loss of function in patient-derived primary glioblastoma stem cells results in a nuclear to cytoplasmic shift of Norrin distribution, and a complete abrogation of its function in stimulating Notch signaling and cellular proliferation.
All-Trans Retinoic Acid Induces Differentiation and Downregulates Stemness Markers and MGMT Expression in Glioblastoma Stem Cells [0.03%]
全反式维甲酸诱导胶质母细胞瘤干细胞分化并下调干性标记和MGMT表达
Justin Tang,Raymond Yang
Justin Tang
Background: Glioblastoma (GBM) remains almost uniformly fatal, owing in part to therapy-resistant cancer stem-like cells (CSCs) and to temozolomide (TMZ) resistance driven by O6-methylguanine-DNA methyltransferase (MGMT)....
Targeting Glioblastoma Stem Cells via EphA2: Structural Insights into the RNA Aptamer A40s for Precision Therapy [0.03%]
靶向胶质母细胞瘤干细胞的EphA2抑制剂RNA适配体A40s结构研究及精准治疗潜力分析
Isidora Diakogiannaki,Vincenzo Maria D&#x;Amore,Alessandra Affinito et al.
Isidora Diakogiannaki et al.
EphA2 receptor tyrosine kinase is overexpressed in many solid tumors and serves as a key driver of tumorigenesis and metastasis. It is highly expressed in glioblastoma multiforme, the most aggressive brain tumor in adults, and in its stem c...
Preclinical approach of two novel tetrahydroquinoline derivatives targeting GPER and Bcl-2 for anti-glioblastoma therapy [0.03%]
两种新型四氢异喹啉衍生物的抗胶质母细胞瘤作用及机制研究
David Méndez-Luna,Loreley-Araceli Morelos-Garnica,José-Rubén García-Sánchez et al.
David Méndez-Luna et al.
G-15 and L-37 to a greater extent than L-06, inhibit neurospheres formation in glioblastoma stem cells (Gli4) cultured in a proliferation medium. Moreover, in 3D antiproliferative assays using glioblastoma stem cells on non-aligned nanofibers L-37 showed better inhibitory effect than L-06....Interestingly, L-06 than L-37 exhibited an antimigratory effect on glioblastoma stem cells loaded onto aligned nanofibers at concentrations where no antiproliferative activity were observed, unlike G-15, a poorly water soluble GPER antagonist.
Androgen receptor inhibition sensitizes glioblastoma stem cells to temozolomide by the miR-1/miR-26a-1/miR-487b signature mediated WT1 and FOXA1 silencing [0.03%]
雄激素受体抑制通过WT1和FOXA1沉默的miR-1 / miR-26a-1 / miR-487b信号使胶质母细胞瘤干细胞对替莫唑胺敏感化
Ana Belén Díaz Méndez,Marta Di Giuliani,Andrea Sacconi et al.
Ana Belén Díaz Méndez et al.
Glioblastomas (GBMs) are aggressive brain tumors and challenging cancers for diagnosis and treatment. Therapeutic options include surgery followed by chemotherapy with the DNA alkylator temozolomide (TMZ) and radiotherapy. However, the pati...
Tumor heterogeneity and resistance in glioblastoma: the role of stem cells [0.03%]
胶质母细胞瘤的肿瘤异质性和耐药性：干细胞的作用
Nikita Ghosh,Debarpan Chatterjee,Aparna Datta
Nikita Ghosh
Glioblastoma multiforme (GBM) is one of the most aggressive and treatment-resistant brain tumor, characterized by its heterogeneity and the presence of glioblastoma stem cells (GSCs).
USP18 deubiquitinates and stabilizes SOX9 to promote the stemness and malignant progression of glioblastoma [0.03%]
USP18通过去泛素化和稳定SOX9促进胶质母细胞瘤干细胞特性及恶性进展
Zhiyuan Liu,Kuo Yu,Kaile Chen et al.
Zhiyuan Liu et al.
Glioblastoma (GBM), the most common and aggressive primary brain tumour, is associated with poor prognosis, primarily due to its stem-like subpopulation, glioblastoma stem cells (GSCs).
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