SLAMF7 Restrains Pro-Inflammatory Macrophage Activation to Counteract Doxorubicin-Induced Cardiotoxicity [0.03%]
SLAMF7通过抑制促炎性巨噬细胞活化来对抗多柔比星致心肌毒性
Ao Liu,Peiyuan Bai,Hongmin You et al.
Ao Liu et al.
Doxorubicin-induced cardiotoxicity (DIC) poses a significant challenge in cancer treatment. This study investigated the role of SLAMF7 in DIC, particularly in macrophage-mediated inflammation. Using SLAMF7 knockout mice, we found that SLAMF...
Pro-inflammatory macrophage activation does not require inhibition of oxidative phosphorylation [0.03%]
促炎症巨噬细胞活化不需要氧化磷酸化的抑制
Andréa B Ball,Anthony E Jones,Kaitlyn B Nguyễn et al.
Andréa B Ball et al.
Pro-inflammatory macrophage activation is a hallmark example of how mitochondria serve as signaling organelles. Oxidative phosphorylation sharply decreases upon classical macrophage activation, as mitochondria are thought to shift from ATP ...
Pro-inflammatory macrophage activation does not require inhibition of mitochondrial respiration [0.03%]
促炎巨噬细胞活化不需要抑制线粒体呼吸
Andréa B Ball,Anthony E Jones,Kaitlyn B Nguyễn et al.
Andréa B Ball et al.
Pro-inflammatory macrophage activation is a hallmark example of how mitochondria serve as signaling organelles. Upon classical macrophage activation, oxidative phosphorylation sharply decreases and mitochondria are repurposed to accumulate ...
Vascular endothelial-derived SPARCL1 exacerbates viral pneumonia through pro-inflammatory macrophage activation [0.03%]
血管内皮源性SPARCL1通过促炎巨噬细胞活化加重病毒性肺炎
Gan Zhao,Maria E Gentile,Lulu Xue et al.
Gan Zhao et al.
Inflammation induced by lung infection is a double-edged sword, moderating both anti-viral and immune pathogenesis effects; the mechanism of the latter is not fully understood. Previous studies suggest the vasculature is involved in tissue ...
SEPTIN2 suppresses an IFN-γ-independent, proinflammatory macrophage activation pathway [0.03%]
SEPTIN2抑制独立于IFN-γ的促炎性巨噬细胞激活途径
Beibei Fu,Yan Xiong,Zhou Sha et al.
Beibei Fu et al.
Interferon-gamma (IFN-γ) signaling is necessary for the proinflammatory activation of macrophages but IFN-γ-independent pathways, for which the initiating stimuli and downstream mechanisms are lesser known, also contribute. Here we identi...
Correction to: Cigarette smoke exposed airway epithelial cells-derived EVs promote pro-inflammatory macrophage activation in alpha-1 antitrypsin deficiency [0.03%]
Correction to:支气管上皮细胞外囊泡促进α-1抗胰蛋白酶缺乏症中促炎巨噬细胞活化
Nazli Khodayari,Regina Oshins,Borna Mehrad et al.
Nazli Khodayari et al.
Published Erratum
Respiratory research. 2023 Nov 4;24(1):266. DOI:10.1186/s12931-023-02571-7 2023
Vascular Endothelial-derived SPARCL1 Exacerbates Viral Pneumonia Through Pro-Inflammatory Macrophage Activation [0.03%]
血管内皮源性SPARCL1通过促炎巨噬细胞活化加重病毒性肺炎
Gan Zhao,Maria E Gentile,Lulu Xue et al.
Gan Zhao et al.
Inflammation upon infectious lung injury is a double-edged sword: while tissue-infiltrating immune cells and cytokines are necessary to control infection, these same factors often aggravate injury. Full appreciation of both the sources and ...
Sphingosine-1-Phosphate Receptor 4 Attenuates Neutrophilic Airway Inflammation in Experimental Asthma via Repressing Proinflammatory Macrophage Activation [0.03%]
鞘氨醇-1-磷酸受体4通过抑制促炎性巨噬细胞活化来缓解实验性哮喘中的中性粒细胞性气道炎症
Shanshan Wang,Zhen Tian,Yanjiao Lu et al.
Shanshan Wang et al.
Patients with eosinophilic asthma react well to conventional treatment of asthma while individualized therapy for non-eosinophilic endotypes have yet to be developed. Dysregulated sphingosine metabolites are associated with the pathophysiol...
Erratum to microRNA-155 inhibition attenuates myocardial infarction-induced connexin 43 degradation in cardiomyocytes by reducing pro-inflammatory macrophage activation [0.03%]
microRNA-155抑制通过减少促炎巨噬细胞活化减弱心肌梗死诱导的心肌细胞连接蛋白43降解的纠错
Editorial Office
Editorial Office
[This corrects the article DOI: 10.21037/cdt-21-743.]. 2022 Cardiovascular Diagnosis and Therapy. All rights reserved.
Published Erratum
Cardiovascular diagnosis and therapy. 2022 Oct;12(5):756-757. DOI:10.21037/cdt-2022-1 2022
Jae-Su Moon,Flavia Franco da Cunha,Jin Young Huh et al.
Jae-Su Moon et al.
Macrophage proinflammatory activation is an important etiologic component of the development of insulin resistance and metabolic dysfunction in obesity. However, the underlying mechanisms are not clearly understood. Here, we demonstrate tha...
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