Fosl2/c-Jun dimer induces oxidative stress and endoplasmic reticulum stress by enhancing Lcn2 transcription to promote ischemic cerebral infarction
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Objective: Ischemic cerebral infarction (ICI) results in high disability and mortality rates. This study aims to validate that the Fosl2/c-Jun dimer promotes ICI via the transcriptional activation of Lcn2. ... ...
