Tapt1 deficiency in mice impairs pulmonary lipid homeostasis and normal postnatal respiration by targeting ABCA3 for autophagy-lysosomal degradation
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TAPT1, which encodes a highly conserved multi-pass transmembrane protein termed transmembrane anterior posterior transformation 1 (TAPT1), has been reported as a disease-causing gene, but its physiological role in mice remains to be elucidated. Using Tapt1 kno... ...
