SMARCA4 activation engages FOSL1 to drive enhancer reprogramming and tumorigenic phenotypes in SMARCA4-deficient LUAD cells
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SMARCA4, the ATPase component of the SWI/SNF chromatin remodeling complex, is integral to the regulation of gene expression through modulation of chromatin accessibility. Although SMARCA4 is frequently inactivated in lung adenocarcinoma (LUAD), a subset of tum... ...
