Nose-to-brain delivery of a SOD1-stabilizing small molecule ameliorates pathology in an ALS mouse model
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Exposure of a pathogenic β6/β7 loop neo-epitope has been proposed to contribute to the pathogenesis of misfolded Cu/Zn superoxide dismutase (SOD1) in amyotrophic lateral sclerosis (ALS) by mediating early events in its noxious structural transformation and p... ...
