Gallic Acid as a Non-cytotoxic Modulator of the ROS-EGFR-ERK-EMT Axis in Xenograft-derived T98G Glioblastoma Cells
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Background/aim: Glioblastoma (GBM) cells that adapt to the in vivo microenvironment often display enhanced invasion driven by EGFR-ERK signaling and epithelial-mesenchymal transition (EMT). This study aimed to characterize an EGF... ...
