LIN28A-Dependent Kinome and Phosphoproteome Reprogramming Promotes Imatinib Resistance
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Chronic myeloid leukemia (CML) resistance to BCR-ABL tyrosine kinase inhibitors (TKIs) can arise from ABL kinase domain mutations, BCR-ABL fusion gene amplification, or kinase-independent mechanisms. To investigate imatinib-resistance, we performed quantitativ... ...
