Nuclear accumulated µ-calpain enhances FoxO3a phosphorylation in AT2 cell and exacerbates pulmonary fibrosis
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Background: µ-calpain is implicated in idiopathic pulmonary fibrosis (IPF), however its role in the aberrant alveolar epithelial type II (AT2) cells differentiation, and its relationship with FoxO3a, an important transcription f... ...
