Dietary K+ supplementation restores normal aldosterone level in Na+-deprived renal tubule-specific CAP1/Prss8-deficient mice
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We previously reported that Na+-deprived mice lacking CAP1/Prss8 in kidney tubules maintained ENaC-mediated sodium balance albeit persistent hypoaldosteronism, hence indicating an uncoupling from aldosterone production. This further suggested an implication of... ...
