OTOP3 functions as an oncogenic regulator of ferroptosis-mediated colorectal cancer progression
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Background: OTOP3, a proton channel located at 17q25.1, drives colorectal cancer growth by enhancing c-Myc stability and inhibiting ferroptosis through GPX4 regulation. Inhibition of OTOP3 suppresses CRC proliferation via c-Myc d... ...
