miR-26a improves microglial activation and neuronal apoptosis in a rat model of cerebral infarction by regulating the TREM1-TLR4/MyD88/NF-κB axis
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Emerging studies have indicated that abnormally expressed microRNAs (miRNAs) are related to the pathogenesis of cerebral ischemia. Nevertheless, the function of miR-26a in neuronal damage and microglial activation during cerebral infarction remains elusive. It... ...
