A peptide-based inhibitor of gp96 suppresses HBsAg expression and HBV replication by upregulation of p53
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In hepatitis B virus (HBV) infection, the virus produces redundant hepatitis B surface antigen (HBsAg) that plays a key role in driving T-cell tolerance and viral persistence. However, currently available anti-HBV agents have no direct effect on HBsAg transcri... ...
